Molecules and Cells

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Fig. 1.

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Fig. 1. (A) RIN4 negatively regulates PAMP-triggered immunity. (B) PAMP perception by PRR and PRPs increases FLS2-dependent RIN4 phosphorylation at the S141 residue and contributes to PTI. (C) In the absence of RPM1, two effector proteins, AvrB and AvrRpm1, induce RIN4T166 phosphorylation, which is epistatic to RIN4S141 phosphorylation, and repress PTI activation. (D) Activation and inactivation of AHA1 and AHA2 control stomata opening and closing. (E) RIN4 binds to the C-terminal region of H+-ATPase and activates it. The effectors AvrRpm1 and AvrB phosphorylate RIN4T166, resulting in increased association of RIN4 with H+-ATPase and activation of H+-ATPase. AvrRpt2: , AvrB: , AvrRpm1: , RIPK: .
Mol. Cells 2019;42:503~511
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